a172 cell line Search Results


a-172  (ATCC)
99
ATCC a-172
A 172, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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a172 cell line  (Korean Cell Line Bank)
90
Korean Cell Line Bank a172 cell line
A172 Cell Line, supplied by Korean Cell Line Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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transfection reagent molar ratio of plasmids transfected* a-172 cell line kit v; program u-029; nucleofector 2b  (Amaxa)
90
Amaxa transfection reagent molar ratio of plasmids transfected* a-172 cell line kit v; program u-029; nucleofector 2b
Transfection Reagent Molar Ratio Of Plasmids Transfected* A 172 Cell Line Kit V; Program U 029; Nucleofector 2b, supplied by Amaxa, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
transfection reagent molar ratio of plasmids transfected* a-172 cell line kit v; program u-029; nucleofector 2b - by Bioz Stars, 2026-03
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cell line a172  (Multiplexion GmbH)
90
Multiplexion GmbH cell line a172
Cell Line A172, supplied by Multiplexion GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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cell line a172 - by Bioz Stars, 2026-03
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a172 glioma cell line  (SAS institute)
90
SAS institute a172 glioma cell line
A172 Glioma Cell Line, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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a172 glioma cell line - by Bioz Stars, 2026-03
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human astrocytoma cell line a172  (Takeda)
90
Takeda human astrocytoma cell line a172
Human Astrocytoma Cell Line A172, supplied by Takeda, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human astrocytoma cell line a172 - by Bioz Stars, 2026-03
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gbm cell line a 172  (JCRB Cell Bank)
90
JCRB Cell Bank gbm cell line a 172
Suppression of cancer cell proliferation by AMF at 227 kHz for more than 30 min. (A) The effect of different frequencies (kHz) of AMF (250 Amrs) on the proliferation of GB cell lines (U87 and LN229). XTT cell proliferation assays were conducted at various AMF frequencies (kHz) for 30 min, with evaluation occurring 24 h post‐AMF exposure ( n = 4, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 kHz). (B) The impact of varying electric current intensities (Arms) in AMF (227 kHz) on the proliferation of GBM cell lines (U87 and LN229) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 Arms). (C) The effect of different exposure durations (min) to AMF (227 kHz, 250 Amrs) on the proliferation of GBM cell lines (LN229, <t>U251)</t> ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 min). (D) The influence of AMF (227 kHz, 250 Amrs) on other GB cell lines (U251, T98, and A172), a pancreatic cell line (PANC1), human breast cancer cell lines (MCF7, MDA‐MB‐231, MDA‐MB‐453), normal human astrocyte (NHA), human cardiac fibroblast (HCF), and human umbilical vein endothelial cells (HUVEC) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. CTRL). (E, F) Continuous monitoring of cell growth with and without a 30‐min AMF exposure (227 kHz, 250 Arms) in U251 and LN229 cell lines. In vitro cell proliferation was measured using the xCELLigence Real‐Time Cellular Analysis system. (G) Cell cycle analysis 3 and 24 h post‐AMF exposure (227 kHz, 250 Arms, 30 min), revealing the inhibitory effect of AMF, notably the induction of S and G2 phase arrest ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL). (H) Immunoblot analysis of phosphorylated and unphosphorylated forms of p53, p21, CDK2, Cyclin A, Cyclin B1, Cyclin D1, Cyclin E, and GAPDH 24 h after a 30‐min AMF exposure (227 kHz, 250 Arms) ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL).
Gbm Cell Line A 172, supplied by JCRB Cell Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/gbm cell line a 172/product/JCRB Cell Bank
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gbm cell line a 172 - by Bioz Stars, 2026-03
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glioblastoma cell line a172  (Wolters Kluwer Health)
90
Wolters Kluwer Health glioblastoma cell line a172
Suppression of cancer cell proliferation by AMF at 227 kHz for more than 30 min. (A) The effect of different frequencies (kHz) of AMF (250 Amrs) on the proliferation of GB cell lines (U87 and LN229). XTT cell proliferation assays were conducted at various AMF frequencies (kHz) for 30 min, with evaluation occurring 24 h post‐AMF exposure ( n = 4, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 kHz). (B) The impact of varying electric current intensities (Arms) in AMF (227 kHz) on the proliferation of GBM cell lines (U87 and LN229) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 Arms). (C) The effect of different exposure durations (min) to AMF (227 kHz, 250 Amrs) on the proliferation of GBM cell lines (LN229, <t>U251)</t> ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 min). (D) The influence of AMF (227 kHz, 250 Amrs) on other GB cell lines (U251, T98, and A172), a pancreatic cell line (PANC1), human breast cancer cell lines (MCF7, MDA‐MB‐231, MDA‐MB‐453), normal human astrocyte (NHA), human cardiac fibroblast (HCF), and human umbilical vein endothelial cells (HUVEC) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. CTRL). (E, F) Continuous monitoring of cell growth with and without a 30‐min AMF exposure (227 kHz, 250 Arms) in U251 and LN229 cell lines. In vitro cell proliferation was measured using the xCELLigence Real‐Time Cellular Analysis system. (G) Cell cycle analysis 3 and 24 h post‐AMF exposure (227 kHz, 250 Arms, 30 min), revealing the inhibitory effect of AMF, notably the induction of S and G2 phase arrest ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL). (H) Immunoblot analysis of phosphorylated and unphosphorylated forms of p53, p21, CDK2, Cyclin A, Cyclin B1, Cyclin D1, Cyclin E, and GAPDH 24 h after a 30‐min AMF exposure (227 kHz, 250 Arms) ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL).
Glioblastoma Cell Line A172, supplied by Wolters Kluwer Health, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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glioblastoma cell line a172 - by Bioz Stars, 2026-03
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human glioblastoma cell line a172-derived dna  (BioChain Institute)
90
BioChain Institute human glioblastoma cell line a172-derived dna
Suppression of cancer cell proliferation by AMF at 227 kHz for more than 30 min. (A) The effect of different frequencies (kHz) of AMF (250 Amrs) on the proliferation of GB cell lines (U87 and LN229). XTT cell proliferation assays were conducted at various AMF frequencies (kHz) for 30 min, with evaluation occurring 24 h post‐AMF exposure ( n = 4, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 kHz). (B) The impact of varying electric current intensities (Arms) in AMF (227 kHz) on the proliferation of GBM cell lines (U87 and LN229) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 Arms). (C) The effect of different exposure durations (min) to AMF (227 kHz, 250 Amrs) on the proliferation of GBM cell lines (LN229, <t>U251)</t> ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 min). (D) The influence of AMF (227 kHz, 250 Amrs) on other GB cell lines (U251, T98, and A172), a pancreatic cell line (PANC1), human breast cancer cell lines (MCF7, MDA‐MB‐231, MDA‐MB‐453), normal human astrocyte (NHA), human cardiac fibroblast (HCF), and human umbilical vein endothelial cells (HUVEC) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. CTRL). (E, F) Continuous monitoring of cell growth with and without a 30‐min AMF exposure (227 kHz, 250 Arms) in U251 and LN229 cell lines. In vitro cell proliferation was measured using the xCELLigence Real‐Time Cellular Analysis system. (G) Cell cycle analysis 3 and 24 h post‐AMF exposure (227 kHz, 250 Arms, 30 min), revealing the inhibitory effect of AMF, notably the induction of S and G2 phase arrest ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL). (H) Immunoblot analysis of phosphorylated and unphosphorylated forms of p53, p21, CDK2, Cyclin A, Cyclin B1, Cyclin D1, Cyclin E, and GAPDH 24 h after a 30‐min AMF exposure (227 kHz, 250 Arms) ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL).
Human Glioblastoma Cell Line A172 Derived Dna, supplied by BioChain Institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/human glioblastoma cell line a172-derived dna/product/BioChain Institute
Average 90 stars, based on 1 article reviews
human glioblastoma cell line a172-derived dna - by Bioz Stars, 2026-03
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Image Search Results


Suppression of cancer cell proliferation by AMF at 227 kHz for more than 30 min. (A) The effect of different frequencies (kHz) of AMF (250 Amrs) on the proliferation of GB cell lines (U87 and LN229). XTT cell proliferation assays were conducted at various AMF frequencies (kHz) for 30 min, with evaluation occurring 24 h post‐AMF exposure ( n = 4, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 kHz). (B) The impact of varying electric current intensities (Arms) in AMF (227 kHz) on the proliferation of GBM cell lines (U87 and LN229) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 Arms). (C) The effect of different exposure durations (min) to AMF (227 kHz, 250 Amrs) on the proliferation of GBM cell lines (LN229, U251) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 min). (D) The influence of AMF (227 kHz, 250 Amrs) on other GB cell lines (U251, T98, and A172), a pancreatic cell line (PANC1), human breast cancer cell lines (MCF7, MDA‐MB‐231, MDA‐MB‐453), normal human astrocyte (NHA), human cardiac fibroblast (HCF), and human umbilical vein endothelial cells (HUVEC) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. CTRL). (E, F) Continuous monitoring of cell growth with and without a 30‐min AMF exposure (227 kHz, 250 Arms) in U251 and LN229 cell lines. In vitro cell proliferation was measured using the xCELLigence Real‐Time Cellular Analysis system. (G) Cell cycle analysis 3 and 24 h post‐AMF exposure (227 kHz, 250 Arms, 30 min), revealing the inhibitory effect of AMF, notably the induction of S and G2 phase arrest ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL). (H) Immunoblot analysis of phosphorylated and unphosphorylated forms of p53, p21, CDK2, Cyclin A, Cyclin B1, Cyclin D1, Cyclin E, and GAPDH 24 h after a 30‐min AMF exposure (227 kHz, 250 Arms) ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL).

Journal: Cancer Science

Article Title: Alternative magnetic field exposure suppresses tumor growth via metabolic reprogramming

doi: 10.1111/cas.16243

Figure Lengend Snippet: Suppression of cancer cell proliferation by AMF at 227 kHz for more than 30 min. (A) The effect of different frequencies (kHz) of AMF (250 Amrs) on the proliferation of GB cell lines (U87 and LN229). XTT cell proliferation assays were conducted at various AMF frequencies (kHz) for 30 min, with evaluation occurring 24 h post‐AMF exposure ( n = 4, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 kHz). (B) The impact of varying electric current intensities (Arms) in AMF (227 kHz) on the proliferation of GBM cell lines (U87 and LN229) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 Arms). (C) The effect of different exposure durations (min) to AMF (227 kHz, 250 Amrs) on the proliferation of GBM cell lines (LN229, U251) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. 0 min). (D) The influence of AMF (227 kHz, 250 Amrs) on other GB cell lines (U251, T98, and A172), a pancreatic cell line (PANC1), human breast cancer cell lines (MCF7, MDA‐MB‐231, MDA‐MB‐453), normal human astrocyte (NHA), human cardiac fibroblast (HCF), and human umbilical vein endothelial cells (HUVEC) ( n = 4, ns, not significant, * p < 0.05, ** p < 0.01, *** p < 0.001 vs. CTRL). (E, F) Continuous monitoring of cell growth with and without a 30‐min AMF exposure (227 kHz, 250 Arms) in U251 and LN229 cell lines. In vitro cell proliferation was measured using the xCELLigence Real‐Time Cellular Analysis system. (G) Cell cycle analysis 3 and 24 h post‐AMF exposure (227 kHz, 250 Arms, 30 min), revealing the inhibitory effect of AMF, notably the induction of S and G2 phase arrest ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL). (H) Immunoblot analysis of phosphorylated and unphosphorylated forms of p53, p21, CDK2, Cyclin A, Cyclin B1, Cyclin D1, Cyclin E, and GAPDH 24 h after a 30‐min AMF exposure (227 kHz, 250 Arms) ( n = 4, ns, not significant, ** p < 0.01, *** p < 0.001 vs. CTRL).

Article Snippet: Human GBM cell lines, U251 MG‐Luc (U251, JCRB1386) and A‐172 (A172, JCRB0228) were purchased from the Japanese Collection of Research Bioresources (JCRB) Cell Bank.

Techniques: In Vitro, Cell Cycle Assay, Western Blot